How to Raise Your Resting Heart Rate When It’s Too Low

A resting heart rate below 60 beats per minute is technically called bradycardia, but it isn’t always a problem. If your heart rate sits between 40 and 60 bpm and you feel fine, there’s generally no reason for concern. The goal isn’t to hit a specific number. It’s to figure out why your heart rate is low and whether that’s causing symptoms like dizziness, fatigue, fainting, or shortness of breath. If it is, raising it means addressing the underlying cause.

When a Low Heart Rate Actually Needs Attention

Normal resting heart rate for adults falls between 60 and 100 bpm, but “normal” is a wide range. Population studies often use 50 bpm as the threshold where low heart rate becomes clinically meaningful, and current cardiology guidelines define concerning slowness as a sustained rate below 50 bpm or pauses longer than 3 seconds between beats.

A heart rate below 40 bpm that’s unusual for you warrants emergency attention, especially if it comes with lightheadedness, confusion, chest discomfort, or signs of poor circulation like cold hands and feet. Without those symptoms, a slow heart rate is typically something to monitor rather than treat aggressively.

Athletic Heart Rate vs. a Problem

If you exercise regularly, especially endurance activities like running, cycling, or swimming, your low heart rate may simply reflect a well-conditioned heart. Up to 80% of endurance athletes develop resting heart rates below 60 bpm. In a study of 465 endurance athletes published in Circulation, 38% had minimum heart rates at or below 40 bpm on a 24-hour monitor, and the vast majority tolerated it without any issues.

Your heart adapts to sustained aerobic training by pumping more blood per beat, so it doesn’t need to beat as often to keep up. This is a sign of cardiovascular efficiency, not disease. Current guidelines from the American College of Cardiology recommend reassurance for any degree of slow heart rate in the absence of symptoms or structural heart problems. The only flag for athletes: a resting rate below 30 bpm may warrant further evaluation regardless of how you feel, though even that hasn’t been clearly linked to adverse events.

Medications That Slow Your Heart

Beta-blockers are the most common medication-related cause of a low heart rate. They work by blocking the stress hormones that speed up your heart, and slowing your pulse is their intended effect. Metoprolol is the most widely prescribed, but the entire class, including propranolol, atenolol, carvedilol, and others, can lower your resting rate significantly.

If your heart rate has dropped since starting a beta-blocker (or a calcium channel blocker, or certain heart rhythm medications), the fix may be as straightforward as adjusting your dose. Your prescriber can taper the medication or switch to an alternative. Never stop a beta-blocker abruptly, since that can cause a dangerous rebound spike in heart rate and blood pressure.

Thyroid and Metabolic Causes

An underactive thyroid is one of the most overlooked reasons for a persistently slow heart rate. Thyroid hormones directly influence how fast your heart beats and how elastic your blood vessels are. When thyroid levels drop, your heart rate slows, your blood pressure can rise (because stiffer arteries require more force to move blood), and your overall metabolism downshifts. You might also notice weight gain, cold intolerance, dry skin, and fatigue.

A simple blood test can confirm hypothyroidism. Treating it with thyroid hormone replacement typically brings your heart rate back into a normal range as your metabolism normalizes. This is one of the most fixable causes of bradycardia.

Electrolyte Imbalances That Affect Heart Rate

Your heart’s electrical system depends on the right balance of minerals in your blood, and imbalances can slow or disrupt your heart rhythm in specific ways.

  • High potassium is a well-known cause of slowed heart conduction. As levels rise, the electrical signals that coordinate each heartbeat become sluggish, potentially progressing to dangerously slow rhythms or cardiac arrest if untreated.
  • High magnesium can depress heart rate directly. Extremely elevated levels produce bradycardia along with drowsiness and slowed breathing.
  • High calcium above certain thresholds can decrease the heart’s ability to generate its own rhythm and may cause progressive conduction delays.

These imbalances are usually identified through routine blood work. Kidney disease, certain medications, and supplements taken in excess are common culprits. Correcting the imbalance corrects the heart rate.

Overtraining and Nervous System Fatigue

There’s a point where more exercise stops helping and starts creating problems. Your heart rate is controlled by two branches of your nervous system: one that speeds it up (sympathetic) and one that slows it down (parasympathetic). Athletes who push through repeated high-intensity training cycles without adequate recovery can develop cumulative autonomic fatigue. This can look like an unusually low resting heart rate paired with persistent tiredness, poor performance, and slow recovery between sessions.

If you’re an athlete and your resting heart rate has dropped below your personal baseline alongside symptoms of burnout, the solution is structured rest. Reducing training volume and intensity for several weeks allows your nervous system to recalibrate. Tracking your resting heart rate each morning can help you spot the trend and confirm recovery.

Practical Steps to Address a Low Heart Rate

Because a low resting heart rate is almost always a symptom rather than a standalone condition, raising it means identifying and treating the cause. Here’s what that looks like in practice:

  • Review your medications. If you take beta-blockers, calcium channel blockers, or antiarrhythmic drugs, ask your prescriber whether a dose adjustment could help. This is the single most actionable step for many people.
  • Get blood work. Thyroid function, potassium, magnesium, and calcium levels can all be checked with standard lab tests. Abnormalities in any of these are treatable.
  • Assess your training load. If you exercise heavily and feel unusually fatigued, consider whether overtraining might be a factor. A planned recovery period is both diagnostic and therapeutic.
  • Reduce vagal triggers. Straining during bowel movements, bearing down, or even tight collars can stimulate the vagus nerve and temporarily slow heart rate. If you notice your heart rate drops during specific activities, avoiding those triggers can help.
  • Stay hydrated and nourished. Dehydration and caloric restriction (especially extreme dieting) can both contribute to a slower heart rate by reducing overall metabolic demand.

When Medical Intervention Is Needed

For people whose slow heart rate causes significant symptoms and doesn’t respond to correcting an underlying cause, a pacemaker is the definitive treatment. A pacemaker is a small device implanted under the skin near the collarbone that sends electrical signals to your heart when it detects the rate has dropped too low. It essentially sets a floor for your heart rate.

Pacemaker implantation is a common procedure, typically done under local anesthesia with sedation, and most people go home the same day or the next morning. Recovery takes a few weeks of limiting arm movement on the implant side, after which most people return to normal activity. Modern pacemakers last 10 to 15 years before the battery needs replacement.

In acute situations where heart rate drops dangerously low and causes symptoms like fainting or confusion, hospitals can use medications or temporary external pacing to raise the rate while the underlying problem is sorted out. These are short-term bridges, not long-term solutions.