What Causes Eating Disorders in Teens: Risk Factors

Eating disorders in teenagers arise from a collision of genetic vulnerability, brain chemistry, hormonal changes during puberty, and environmental pressures. No single factor is enough on its own. Between 2017 and 2022, diagnosed eating disorders among 13- to 18-year-olds nearly quintupled, rising from 112 to 560 per 100,000, with the sharpest increase among girls in that age group. Understanding what drives these disorders means looking at biology, psychology, and the social world teens inhabit, because all three interact.

Genetics Set the Stage

Eating disorders run in families, and twin studies make it clear that a large share of the risk is inherited. Heritability estimates for anorexia nervosa range from 48% to 74%, meaning roughly half to three-quarters of the variation in risk can be traced to genetic factors. For bulimia nervosa, heritability falls between 55% and 62%. Binge eating disorder has a somewhat lower but still meaningful genetic contribution, estimated at 39% to 45%.

These numbers don’t mean a specific gene causes an eating disorder. Instead, many genes each contribute a small amount of risk, influencing things like how the brain processes reward, how a teen responds to stress, and how sensitive they are to hunger and fullness signals. A teenager can carry significant genetic risk and never develop an eating disorder if the right environmental triggers never arrive. But when they do, that genetic load makes the difference between a teen who brushes off a diet culture message and one who spirals into restriction or bingeing.

Puberty Activates Hidden Risk

One of the most striking findings in eating disorder research is that genetic risk appears to be dormant before puberty and switches on during it. Twin studies tracking girls across adolescence found essentially 0% heritability for eating disorder symptoms at age 11, but over 50% heritability by age 17. The timing closely tracks the rise in estrogen during puberty.

Estrogen and other hormones released during puberty don’t just change the body’s shape. They regulate gene activity in the brain, influencing the production of chemical messengers tied to mood, appetite, and reward. Researchers now believe that rising estrogen levels may flip on susceptibility genes that were silent in childhood. This helps explain why the typical onset age for anorexia is around 16 to 19, and for bulimia, 14 to 20. The hormonal surge of puberty is not just a background event. It’s a biological trigger.

This also explains the gender gap. Girls experience a larger and earlier rise in estrogen, which may be why eating disorders are diagnosed at far higher rates in teenage girls than boys. Among females aged 13 to 18, prevalence jumped from 120 per 100,000 in 2017 to 916 per 100,000 in 2022.

Brain Reward Circuits Work Differently

Teens who develop eating disorders often have differences in how their brains respond to food, body image, and reward. The brain’s reward system, which drives the motivation to eat and feel pleasure from food, shows clear alterations in people with eating disorders compared to those without.

In anorexia, the brain’s dopamine-related reward response appears to be hypersensitive. Teens with anorexia show stronger brain responses to unexpected tastes and to images of thin bodies than their peers do. Paradoxically, this heightened sensitivity may make food restriction feel rewarding. Animal research supports this: food restriction and weight loss increase dopamine-related reward activity in the brain, which could reinforce the cycle of not eating.

Bulimia shows a different pattern. Repeated binge-and-purge cycles are associated with a blunted reward response, similar to what researchers see in addiction. The brain’s prediction system becomes less responsive over time, and higher binge-purge frequency predicts even lower brain reactivity. This may drive the need for increasingly larger binges to achieve the same sense of relief or satisfaction.

The insula, a brain region that helps you sense what’s happening inside your body (like stomach fullness or a racing heart), also functions differently. Even after weight restoration, people who had anorexia show altered insula responses to stomach and heart signals, and these differences correlate with anxiety levels and eating disorder severity. When a teen can’t accurately read their own hunger and fullness cues, maintaining normal eating becomes much harder.

Perfectionism and Anxiety as Psychological Fuel

Certain personality traits act as psychological kindling. Perfectionism is consistently elevated in people with eating disorders compared to healthy peers, and it predicts eating disorder development before symptoms even begin. Critically, perfectionism remains elevated even after full recovery, suggesting it’s a pre-existing trait rather than a consequence of the illness.

Anxiety disorders are similarly overrepresented. About 42% of people with anorexia and 38% of those with bulimia meet criteria for a current anxiety disorder, compared to 16% of people without an eating disorder. For a teen who already experiences high anxiety, controlling food intake can become a way to manage overwhelming feelings of uncertainty. The eating disorder provides a false sense of control that temporarily quiets the anxiety, creating a powerful reinforcement loop.

Depression often travels alongside these traits. Teens who are partially recovered from eating disorders are more likely to have a current mood disorder than those who fully recover, suggesting that unresolved depression can keep the door open for relapse.

Social Media and Appearance Comparison

Social media has introduced a uniquely potent environmental trigger. The core mechanism is upward appearance comparison: scrolling through images of people a teen perceives as more attractive. Experimental research shows that these comparisons lead to immediate drops in body satisfaction and increased urges to restrict food. The effect is bidirectional and self-reinforcing. After making upward comparisons, teens feel worse about their bodies, which in turn drives them to make more upward comparisons.

Comparisons to peers hit harder than comparisons to celebrities. When an attractive person feels attainable, like a classmate or someone the same age, the gap between “how I look” and “how they look” feels more personal and more painful. This makes peer-driven social media content particularly damaging. Upward comparisons also predict increased urges to overeat, not just restrict, which means the same social media behavior can push different teens toward opposite but equally harmful patterns.

Family Environment and Weight Talk

The home environment shapes a teen’s relationship with food and body image in ways that can either protect against or contribute to eating disorders. Families with rigid rules, especially rules that restrict emotional expression or tightly control food, create conditions where disordered eating is more likely to take root. When teens aren’t allowed to voice discomfort or negative feelings, those emotions often get channeled into the one thing they can control: eating.

A critical attitude about weight and physical appearance from family members is a particularly strong risk factor. Parental comments about a teen’s body, even well-intentioned ones, family dieting culture, and pressure to look a certain way all predict the development of unhealthy eating habits. For pre-adolescent and adolescent girls, family weight concerns combined with media beauty ideals predict an increased risk of binge eating specifically.

Different Pressures for Boys

Eating disorders in teenage boys are rising but often look different than in girls. While girls more commonly face pressure toward thinness, boys increasingly face pressure toward muscularity and leanness. The “drive for muscularity,” fueled by decades of increasingly muscular male imagery in media, from action figures to fitness influencers, is now recognized as a significant risk pathway.

Social media plays a specific role for boys. Image-based platforms expose them to filtered, unrealistic body ideals, and algorithms continuously serve muscle-related content to anyone who engages with it. Boys report using social media to learn about diets, weight training routines, and supplements, and content related to muscular bodies and muscle-building supplements is associated with body dysmorphia symptoms even after controlling for total time spent online. Traditional masculinity norms that emphasize strength and dominance add another layer of pressure, turning body dissatisfaction into pathological behaviors like extreme dietary restriction, compulsive exercise, or use of performance-enhancing substances.

Boys with eating disorders are also more likely to be diagnosed at younger ages, particularly with avoidant or restrictive eating patterns, which tend to appear in pre-pubertal children rather than during the mid-teen years when anorexia and bulimia typically emerge in girls.

How These Causes Interact

No teen develops an eating disorder because of one factor alone. The typical path involves a genetically vulnerable teen entering puberty, where rising hormones activate dormant genetic risk and reshape brain reward circuitry. If that teen also carries traits like perfectionism or anxiety, they’re primed to respond to environmental triggers: a comment from a parent about weight, a social media feed full of idealized bodies, a peer group where dieting is normal. Each layer compounds the others.

This layered model also explains why two teens in the same environment can have completely different outcomes. The one who develops an eating disorder isn’t weaker or more vain. They’re carrying a biological and psychological load that made them vulnerable in ways that were invisible until the right combination of triggers arrived. Recognizing this complexity is the first step toward catching the warning signs early, when intervention is most effective and recovery is most likely.