Why Do Doctors Check Reflexes During an Exam?

Reflexes, the rapid and involuntary muscle responses to external stimuli, offer a non-invasive way for medical professionals to assess the integrity of the nervous system. Testing these automatic reactions provides immediate insight into whether the sensory and motor pathways are functioning correctly. This examination is particularly valuable because it requires no conscious patient participation, making it a reliable indicator of nervous system health and the pathways that connect the body to the spinal cord and brain.

The Automatic Nature of the Reflex Arc

The speed of a reflex action is possible because the signal does not need to travel to the brain for processing. Instead, the action is controlled by the reflex arc, a dedicated neural pathway that allows for a swift, protective response.

The process begins with a receptor, which detects the external stimulus, such as the sudden stretch of a tendon. This information is then carried toward the spinal cord by an afferent, or sensory, neuron. Once inside the spinal cord, the signal reaches an integration center, which may involve a single direct connection or multiple interneurons.

From the integration center, an efferent, or motor, neuron carries the response signal away from the spinal cord. This motor neuron directly instructs the effector—typically a muscle—to contract, completing the reflex action. By bypassing the brain’s complex analysis centers, the reflex arc ensures an immediate and automatic reaction, demonstrating the functional status of the spinal cord and peripheral nerves.

Key Reflexes Tested During an Exam

Doctors commonly test several categories of reflexes, each providing a window into different levels of the nervous system. Deep Tendon Reflexes (DTRs), sometimes called muscle stretch reflexes, are the most frequently assessed. These are elicited by tapping a tendon, which briefly stretches the attached muscle and triggers a contraction.

Common DTRs include the patellar reflex (knee-jerk), Achilles reflex (ankle-jerk), biceps, triceps, and brachioradialis reflexes. These tests assess specific spinal nerve roots (L2–L4, S1–S2, C5–C6, C7–C8, and C6, respectively). The doctor uses a reflex hammer to strike the tendon briskly, observing the resulting muscle movement.

Another category is superficial reflexes, such as the plantar reflex, which is often called the Babinski test. This reflex is elicited by firmly stroking the sole of the foot with a blunt instrument. The normal adult response is a downward curling of the toes, known as plantar flexion.

Cranial nerve reflexes are also part of the examination, assessing nerve pathways in the head and face. A common example is the pupillary light reflex, where a light source is shone into the eye. A normal response involves the pupils constricting simultaneously, which checks the function of the optic and oculomotor cranial nerves.

Interpreting Reflex Results for Neurological Health

The patient’s response to reflex testing provides clues about the location and type of potential nervous system damage. Reflexes are graded on a scale, but the most telling information comes from responses that are diminished, exaggerated, or abnormal. A diminished or absent response, known as hyporeflexia, indicates a problem within the reflex arc itself.

This hyporeflexia is a hallmark sign of a lower motor neuron (LMN) lesion, meaning damage to the nerve cells in the spinal cord or the peripheral nerves leading to the muscle. Since the signal cannot effectively travel along the sensory or motor pathway, the muscle fails to contract normally. Conditions like peripheral neuropathy, which affects nerves outside the central nervous system, often present with diffuse hyporeflexia.

Conversely, an exaggerated response, called hyperreflexia, points to damage in the upper motor neuron (UMN) system. UMNs originate in the brain and travel down to the spinal cord, acting to regulate and suppress the activity of the LMNs. When the UMN pathway is damaged, this inhibitory control is lost, causing the LMNs to fire excessively and resulting in a hyperactive reflex.

The abnormal Babinski sign in an adult is a specific indicator of UMN damage, often affecting the corticospinal tract. A positive Babinski sign occurs when the big toe extends upward and the other toes fan out upon stimulation of the sole. While this response is normal in infants whose nervous system is still maturing, its presence in an adult suggests a serious central nervous system issue, such as stroke or multiple sclerosis. The combination of hyperreflexia and an abnormal plantar reflex helps doctors localize a lesion to the central nervous system.